To perform an epistasis test, we would cross two mutants with the same phenotype and observe their progeny.

A neomorphic mutation results in an allele that produces a novel protein or causes inappropriate expression resulting in a new phenotype.

$q,$ The wildtype version of one type of gene involved in cancer is called a proto$-$oncogene and the mutant version is called an oncogene.

The wildtype versions of tumor suppressor genes inhibit the cell cycle and gain of function mutations can contribute to cancer progression.

A mutation in a non$-$coding region of a gene will never produce a mutant phenotype.

Most human traits are monogenic, or controlled by a single gene.

$q,$ Dihybrid crosses involving two genes in a biochemical pathway with similar colored intermediates will produce progeny with a phenotypic ratio of $9$ organisms with the wildtype phenotype to $7$ organisms with the mutant phenotype.

In a monogenic cross involving two alleles that are co$-$dominant, the phenotypic ratio does not match the genotypic ratio.

$q,$ In determining blood type, the $I^A$ and $I^B$ alleles are co$-$dominant and the H gene is epistatic to the I gene.

$q,$ Genomic libraries are different than cDNA libraries in that genomic libraries contain non$-$coding DNA, such as promoter and introns.