27 133/2 points DL. suffers from primary adrenal insufficiency caused by damage to the adrenal cortex...

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27 133/2 points DL. suffers from primary adrenal insufficiency caused by damage to the adrenal cortex How would this condition affect levels of: CRH Decrease ACTH EIncrease and cortisol 5 Decrease 2points When blood calcium is low, the kidneys decrease reabsorption of Ca2- into the blood and osteociast activity B increases to helo retum calclum levels to homeostasis. 29 H.P. has a metabolic defect caused by a problem with Thyroid hormone signaling. The physician orders tests for TSH and thyraxine (TH). Results are shown below: H.P's levels 22.0 uU/ml 28 ng/dl Normal levels Serum TSH Serum thyroxine (TH) 05-55uU/ml 46-12 ng/di In follow-up tests, serum was obtained from H.P. and tested for the presence of anti-thyro antibodies The following results were obtained: H.P. Normal Ant-thyroid antibody Positve Negstive HP. is suffering from Hashimoto's thyroiditis The observed levels of serum TH are a result of the antibodies destroying the anterlor pituitary 27 133/2 points DL. suffers from primary adrenal insufficiency caused by damage to the adrenal cortex How would this condition affect levels of: CRH Decrease ACTH EIncrease and cortisol 5 Decrease 2points When blood calcium is low, the kidneys decrease reabsorption of Ca2- into the blood and osteociast activity B increases to helo retum calclum levels to homeostasis. 29 H.P. has a metabolic defect caused by a problem with Thyroid hormone signaling. The physician orders tests for TSH and thyraxine (TH). Results are shown below: H.P's levels 22.0 uU/ml 28 ng/dl Normal levels Serum TSH Serum thyroxine (TH) 05-55uU/ml 46-12 ng/di In follow-up tests, serum was obtained from H.P. and tested for the presence of anti-thyro antibodies The following results were obtained: H.P. Normal Ant-thyroid antibody Positve Negstive HP. is suffering from Hashimoto's thyroiditis The observed levels of serum TH are a result of the antibodies destroying the anterlor pituitary

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27 When the cortex is damaged and doesnt produce enough adrenocortical hormones the condition ... View the full answer